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Recombinant Mouse RANK L/TNFSF11, His (CHO-expressed)
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檢測信息查詢
| 貨號 | 規格 | 貨期 | 庫存 | 價格 | 促銷價 | 訂購 |
| 1308132943-10 μg | 0 | ￥0 | ||||
| 1308132943-50 μg | 0 | ￥0 |
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| 性狀 | Recombinant Mouse RANK L/TNFSF11,His (CHO-expressed) 是來源于 CHO 的 TNF 相關的活化誘導細胞因子。人源和鼠源 RANK L/TNFSF11 的同源性約為 82%。Recombinant Mouse RANK L/TNFSF11,His (CHO-expressed) 誘導 JNK 的激活，增強 T 細胞生長和樹突細胞功能，誘導破骨細胞生成和淋巴結器官生成。
Synonyms rMuRANK L/TNFSF11, His; TRANCE; CD254 Species Mouse Source CHO Accession O35235 Gene ID 21943 Molecular Weight Approximately 28.3 kDa AA Sequence HHHHHHRAQM DPNRISEDST HCFYRILRLH ENADLQDSTL ESEDTLPDSC RRMKQAFQGA VQKELQHIVG PQRFSGAPAM MEGSWLDVAQ RGKPEAQPFA HLTINAASIP SGSHKVTLSS WYHDRGWAKI SNMTLSNGKL RVNQDGFYYL YANICFRHHE TSGSVPTDYL QLMVYVVKTS IKIPSSHNLM KGGSTKNWSG NSEFHFYSIN VGGFFKLRAG EEISIQVSNP SLLDPDQDAT YFGAFKVQDI D Biological Activity The ED50 is 2.44 μg/mL as measured. Appearance Lyophilized powder. Formulation Lyophilized after extensive dialysis against PBS, pH 7.4. Endotoxin Level <1.0 EU/μg, determined by LAL method. Reconstitution Reconstitute the lyophilized recombinant Mouse RANK L/TNFSF11, His (CHO-expressed) (rMuRANK L/TNFSF11, His) to 100 μg/mL using ddH2O. Storage & Stability Lyophilized recombinant Mouse RANK L/TNFSF11, His (CHO-expressed) (rMuRANK L/TNFSF11, His) is stored at -20°C. After reconstitution, it is stable at 4°C for 1 week or -20°C for longer. It is recommended to freeze aliquots at -20°C or -80°C for extended storage. Shipping Room temperature in continental US; may vary elsewhere. Background Receptor activator of NF-κB ligand (RANKL), its cellular receptor, receptor activator of NF-κB (RANK), and the decoy receptor osteoprotegerin (OPG) constitute a novel cytokine system. RANKL produced by osteoblastic lineage cells and activated T lymphocytes is the essential factor for osteoclast formation, fusion, activation, and survival, thus resulting in bone resorption and bone loss. RANKL activates its specific receptor, RANK located on osteoclasts and dendritic cells, and its signaling cascade involves stimulation of the c-jun, NF-κB, and serine/threonine kinase PKB/Akt pathways[1]. |
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